The hidden impact of heat stress
When fetuses are exposed to elevated temperatures during gestation — a condition known as in utero heat stress — piglets may be born lighter, with a compromised gut microbiome, weakened immune function, and a heightened vulnerability to stress later in life. These effects are not always immediately obvious, yet they are essential to recognize and prevent.
Every degree above 25°C impacts performance:
Three critical moments
(Mating to ~Day 14)
During the first two weeks of pregnancy, embryos are developing and implanting in the uterus. Heat stress increases the sow’s core body temperature and alters the uterine environment (blood flow, hormones, and oxidative balance). These changes can reduce embryo survival and increase early embryonic loss, leading to fewer total pigs born.
(~Day 30 to 60)
Between days 30 and 60, major fetal organs and the placenta are developing. Heat stress during this period can reduce placental efficiency and nutrient supply to the fetus. In female fetuses, this stage also overlaps with important phases of ovarian development. Excessive heat exposure may affect the formation and survival of early germ cells (future oocytes), potentially influencing the reproductive capacity of the offspring gilt.
(Final weeks before farrowing)
In late pregnancy, heat stress increases oxidative stress and causes the sow to redirect blood flow toward the skin to dissipate heat. This can reduce blood flow to the uterus and placenta. As a result, oxygen and nutrient delivery to the fetuses may be compromised, increasing the risk of stillbirths, lower birth weight, and weaker piglets at birth.
Preparation starts long before each stage
Heat stress during these phases can reduce embryo survival, fetal development, and birth weights.
How heat stress affects the sow
Metabolic impact
- Lower efficiency of nutrient utilization
- Heightened oxidative stress
- Activation of the immune system — diverting nutrients away from growth and reproduction
Gut integrity
- Reduced mucin production and secretion
- Increased intestinal permeability (“leaky gut”)
- Shifts in gut microbiota, reducing beneficial bacteria
- Favoring opportunistic pathogens
Reproductive function
- Altered puberty onset and estrus expression
- Decreased secretion of key reproductive hormones
- Impaired milk production
- Prolonged farrowing times
Heat stress before birth affects lifetime performance
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Lactation under heat stress
Lactation is the most energy-demanding stage in a sow’s life, and heat stress greatly amplifies this burden. Beyond these productive losses, heat stress disrupts gut integrity by increasing intestinal permeability (“leaky gut”) and elevating systemic inflammation. Combined, these physiological strains heighten vulnerability to disease and substantially increase the risk of sow mortality during the summer months.
Under elevated temperatures, sows reduce feed intake to limit metabolic heat production, which in turn reduces milk yield, slows piglet growth, and prolongs the weaning‑to‑estrus interval.
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